Vitamin D and Inflammatory Bowel Disease

Inflammatory bowel disease — Crohn's disease and ulcerative colitis — sits at the intersection of a disease with a strong latitude gradient, a population with high rates of vitamin D deficiency, and a biology (mucosal immunity, T-cell regulation, antimicrobial peptides) that vitamin D actively modulates.

Deficiency is the norm in IBD

Cross-sectional studies find 60–70% of adults with active Crohn's have 25(OH)D < 20 ng/mL, and 40–50% of ulcerative colitis patients. Reasons stack: reduced intestinal absorption (especially with terminal ileal Crohn's or resection), reduced sun exposure during flares, corticosteroid effects on vitamin D metabolism, and dietary avoidance of dairy.

The latitude and MR evidence

IBD incidence increases sharply with latitude — the Nurses' Health Study found women in the highest 25(OH)D quartile had 46% lower Crohn's incidence than those in the lowest. Mendelian randomisation studies using genetic variants that lower 25(OH)D suggest a causal link with Crohn's specifically, less clearly with ulcerative colitis.

Supplementation trials

Practical management

  1. Everyone with IBD should have 25(OH)D checked at diagnosis and annually.
  2. Target 25(OH)D 30–50 ng/mL (75–125 nmol/L).
  3. Malabsorption often requires larger doses (2,000–5,000 IU/day) than in the general population; some patients need 10,000 IU/day.
  4. Terminal ileal Crohn's, short-bowel syndrome, or post-resection patients may need 25(OH)D (calcifediol) or IM cholecalciferol if oral is inadequate.
  5. Check calcium at higher doses to avoid iatrogenic hypercalcaemia.
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Not medical advice. Vitamin D is an adjunct in IBD — not a substitute for 5-ASA, immunomodulators, biologics, or JAK inhibitors as clinically indicated.

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