Vitamin D and Preterm Birth

Preterm birth (before 37 weeks) is a leading cause of infant mortality and long-term morbidity worldwide. Maternal vitamin D deficiency has been examined as a modifiable risk factor, with observational associations that supplementation trials partly confirm.

Observational associations

Multiple cohort studies find lower maternal 25(OH)D associated with higher rates of preterm birth. A 2015 meta-analysis reported adjusted OR ~1.3 for deficient vs sufficient women. The MUSC study (Wagner 2016) found each 10 ng/mL increase in maternal 25(OH)D was associated with 40% reduction in early preterm delivery (< 32 weeks).

RCT evidence

Mechanism

Vitamin D regulates immune responses at the maternal-fetal interface, modulates inflammation (which is causally linked to premature membrane rupture and preterm labour), and affects placental development. Vitamin D deficiency correlates with bacterial vaginosis and cervicovaginal infection, both preterm-labour risk factors.

Preterm infant vitamin D

Preterm infants have very low body stores of vitamin D at birth (most vitamin D accrual is in the third trimester). Prevention of preterm rickets and metabolic bone disease of prematurity requires:

Practical guide

  1. Preconception and first-trimester: test 25(OH)D and correct to ≥ 30 ng/mL (some obstetricians target 40+).
  2. Continue 1,000–4,000 IU/day cholecalciferol throughout pregnancy. See pregnancy page.
  3. History of previous preterm birth: aggressive vitamin D correction is one of several evidence-based interventions (progesterone, cervical cerclage as indicated).
  4. Postpartum breastfeeding: see maternal 6,400 IU/day option in the infants page.
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Not medical advice. Preterm birth risk factors require specialised obstetric care — vitamin D is one of many considerations in a pregnancy at risk.

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